대한신장학회

Skip Navigation: Skip to contents

KOR
ENG
전체메뉴보기

간행물 검색

현재 페이지 경로

HOME
간행물
간행물 검색

논문분류	춘계학술대회 초록집
제목	Allograft dysfunction by transplant renal artery stenosis due to posttransplantation subcapsular hematoma removal operation
저자	Jin Ho Lee*, Joong-Kyung Kim, Yong Hun Sin, Joon-Seok Oh, Seong-Min Kim, Dong Yeol Lee
출판정보	2016; 2016(1):
키워드	KTP, transplant renal artery stenosis, PTA
초록	Background: Transplant renal artery stenosis(TRAS) is one of etiology of allograft dysfunction. If TRAS was diagnosed early, it can be cured. But, when diagnosis is delayed, TRAS causes parenchymal necrosis. Methods: 40-year-old woman, who has end stage renal disease due to IgA nephropathy, underwent kidney transplantation with her elder sister. HLA typing shows no mismatch. Total ischemic time was 110 min. POD 2, Urine output was decreased sharply. We checked non enhanced abdominal CT(Figure 1), that shows subcapsular hematoma and pelvic cavity hematoma. She underwent secondary surgery for hematoma removal. In operation room, renal upper polar capsulotomy was done, then mash material was sutured that site. Bleeding control was successful, but serum creatinine level was still high. At POD 25, abdomen MR angiography(Figure 2) shows anatomotic site between renal artery and internal iliac artery showing acute angle with focal stenosis, and focal hypoperfusion lesion. Results: Percutaneous transluminal angioplasty(PTA) was implemented. Significant stenosis(>80%) was detected at anastomotic site(Figure 3), then 5mm stent was inserted at stenotic lesion, 5mm balloon angioplasty was done(Figure 4). Renal arterial diameter and blood flow was normalized. At POD 5month, DMSA scan shows multiple focal radioisotope defect, that means graft parenchymal function insufficiency(Figure 5). Conclusion: 8 month after renal transplantation, serum creatinine level was 3.1mg/dl and GFR was 20ml/min. TRAS was cause of parenchymal necrosis and allograft dysfunction. Table: Figures:
원문(PDF)	PDF 원문보기

목록

위로가기